Cardiac arrest: a new cutting edge treatment method from Vygon

This video explains how b-card “Boussignac Cardiac Arrest Resuscitation Device” works.

Vygon, the single-use medical devices group has announced the commercial launch of the b-card (Boussignac Cardiac Arrest Resuscitation Device), a non-invasive ventilation system providing continuous oxygen delivery during CPR (CardioPulmonary Resuscitation). The device ensures dynamic alveolar ventilation without the need to pause chest compressions. Recent international scientific recommendations advocate, when treating a cardiac arrest, chest compressions should not be interrupted in order to ensure continuous blood flow.

However, it is still desirable to provide oxygen. Therefore emergency responders currently alternate between chest compressions and ventilation at a rate of 30 compressions to two rescue breaths.

The new b-card device eliminates the need to stop compressions to ventilate. Connected to a source delivering oxygen at a flow rate of 15L/minute, the b-card generates a virtual valve. This acts as the “heart” of the device, optimizing the pressure created during the chest compression and decompression phases of resuscitation.



Vygon launches b-card, an innovative device that combines continuous chest compressions and dynamic oxygenation for CPR

Study: Evaluation of the Boussignac Cardiac arrest device (B-card) during cardiopulmonary resuscitation in an animal model

Aim of the study

The purpose of this study was to examine continuous oxygen insufflation (COI) in a swine model of cardiac arrest. The primary hypothesis was COI during standard CPR (S-CPR) should result in higher intrathoracic pressure (ITP) during chest compression and lower ITP during decompression versus S-CPR alone. These changes with COI were hypothesized to improve hemodynamics. The second hypothesis was that changes in ITP with S-CPR + COI would result in superior hemodynamics compared with active compression decompression (ACD) + impedance threshold device (ITD) CPR, as this method primarily lowers ITP during chest decompression.

Methods

After 6 min of untreated ventricular fibrillation, S-CPR was initiated in 8 female swine for 4 min, then 3 min of S-CPR + COI, then 3 min of ACD + ITD CPR, then 3 min of S-CPR + COI. ITP and hemodynamics were continuously monitored.
Results

During S-CPR + COI, ITP was always positive during the CPR compression and decompression phases. ITP compression values with S-CPR + COI versus S-CPR alone were 5.5 ± 3 versus 0.2 ± 2 (p < 0.001) and decompression values were 2.8 ± 2 versus −1.3 ± 2 (p < 0.001), respectively. With S-CPR + COI versus ACD + ITD the ITP compression values were 5.5 ± 3 versus 1.5 ± 2 (p < 0.01) and decompression values were 2.8 ± 2 versus −4.7 ± 3 (p < 0.001), respectively.
Conclusion

COI during S-CPR created a continuous positive pressure in the airway during both the compression and decompression phase of CPR. At no point in time did COI generate a negative intrathoracic pressures during CPR in this swine model of cardiac arrest.

Reference: http://www.sciencedirect.com/science/article/pii/S0300957217303295